Health Problems of Newborns



Health Problems of Newborns


Debbie Fraser



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http://evolve.elsevier.com/wong/essentials





Birth Injuries


image Animation—Shoulder Dystocia


image Several factors predispose an infant to birth injuries (Mangurten and Puppala, 2011; Verklan and Lopez, 2011). Maternal factors include uterine dysfunction that leads to prolonged or precipitous labor, preterm or postterm labor, and cephalopelvic disproportion. Injury may result from dystocia caused by fetal macrosomia, multifetal gestation, abnormal or difficult presentation (not caused by maternal uterine or pelvic conditions), and congenital anomalies. Intrapartum events that can result in scalp injury include the use of intrapartum monitoring of fetal heart rate and collection of fetal scalp blood for acid–base assessment. Obstetric birth techniques can cause injury. Forceps birth, vacuum extraction, version and extraction, and cesarean birth are potential contributory factors. Often more than one factor is present, and multiple predisposing factors may be related to a single maternal condition.



Soft Tissue Injury


Various types of soft tissue injury may be sustained during the process of birth, primarily in the form of bruises or abrasions secondary to dystocia. Soft tissue injury usually occurs when there is some degree of disproportion between the presenting part and the maternal pelvis (cephalopelvic disproportion). The use of forceps to facilitate a difficult vertex delivery may produce bruising or abrasion on the sides of the neonate’s face. Petechiae or ecchymoses may be observed on the presenting part after a breech or brow delivery. After a difficult or precipitous delivery, the sudden release of pressure on the head can produce scleral hemorrhages or generalized petechiae over the face and head. Petechiae and ecchymoses may also appear on the head, neck, and face of an infant born with a nuchal cord, giving the infant’s face a cyanotic appearance. A well-defined circle of petechiae and ecchymoses or abrasions may also be seen on the occipital region of the newborn’s head when a vacuum suction cup is applied during delivery. Rarely, lacerations occur during cesarean section.


These traumatic lesions generally fade spontaneously within a few days without treatment. However, petechiae may be a manifestation of an underlying bleeding disorder or a systemic illness such as an infection and should be further evaluated as to their origin. Nursing care is primarily directed toward assessing the injury and providing an explanation and reassurance to the parents.



Head Trauma


Trauma to the head and scalp that occurs during the birth process is usually benign but occasionally results in more serious injury. The injuries that produce serious trauma, such as intracranial hemorrhage and subdural hematoma, are discussed in relation to neurologic disorders in the newborn (see Table 9-9). Skull fractures are discussed in association with other fractures sustained during the birth process. The three most common types of extracranial hemorrhagic injury are caput succedaneum, cephalhematoma, and subgaleal hemorrhage.



Caput Succedaneum


The most commonly observed scalp lesion is caput succedaneum, a vaguely outlined area of edematous tissue situated over the portion of the scalp that presents in a vertex delivery (Fig. 9-1, A). The swelling consists of serum, blood, or both accumulated in the tissues above the bone, and it often extends beyond the bone margins. The swelling may be associated with overlying petechiae or ecchymoses. No specific treatment is needed, and the swelling subsides within a few days.




Cephalhematoma


Infrequently, a cephalhematoma is formed when blood vessels rupture during labor or delivery to produce bleeding into the area between the bone and its periosteum. The injury occurs most often with primiparous delivery and is often associated with forceps delivery and vacuum extraction. Unlike caput succedaneum, the boundaries of the cephalhematoma are sharply demarcated and do not extend beyond the limits of the bone (suture lines) (Fig. 9-1, B). The cephalhematoma may involve one or both parietal bones. The occipital bones are less commonly affected, and the frontal bones are rarely affected. The swelling is usually minimal or absent at birth and increases in size on the second or third day. Blood loss is usually not significant.


No treatment is indicated for uncomplicated cephalhematoma. Most lesions are absorbed within 2 weeks to 3 months. Lesions that result in severe blood loss to the area or that involve an underlying fracture require further evaluation. Hyperbilirubinemia may result during resolution of the hematoma. A local infection can develop and is suspected when a sudden increase in swelling occurs. Parents should be counseled that, in some cases, a small area of calcification may develop and persist.



Subgaleal Hemorrhage


Subgaleal hemorrhage is bleeding into the subgaleal compartment (Fig. 9-1, C). The subgaleal compartment is a potential space that contains loosely arranged connective tissue; it is located beneath the galea aponeurosis, the tendinous sheath that connects the frontal and occipital muscles and forms the inner surface of the scalp. The injury occurs as a result of forces that compress and then drag the head through the pelvic outlet (Verklan and Lopez, 2011). Instrumented delivery, particularly vacuum extraction and forceps delivery, increase the risk of subgaleal hemorrhage. Additional risk factors include prolonged second stage of labor, fetal distress, macrosomia, failed vacuum extraction, and maternal primiparity (Doumouchtsis and Arulkumaran, 2006). The bleeding extends beyond bone, often posteriorly into the neck, and continues after birth, with the potential for serious complications such as anemia or hypovolemic shock.


Early detection of the hemorrhage is vital; serial head circumference measurements and inspection of the back of the neck for increasing edema and a firm mass are essential. A boggy fluctuant mass over the scalp that crosses the suture line and moves as the baby is repositioned is an early sign of subgaleal hemorrhage (Doumouchtsis and Arulkumaran, 2006). Other signs include pallor, tachycardia, and increasing head circumference (Reid, 2007). An early sign of subgaleal hemorrhage is a forward and lateral positioning of the newborn’s ears because the hematoma extends posteriorly. Disseminated intravascular coagulation (DIC) has also been reported in association with subgaleal hemorrhage (Schierholz and Walker, 2010). Computed tomography (CT) or magnetic resonance imaging (MRI) is useful in confirming the diagnosis. Replacement of lost blood and clotting factors is required in acute cases of hemorrhage. Monitoring the infant for changes in level of consciousness and a decrease in the hematocrit are also key to early recognition and management. An increase in serum bilirubin levels may be seen as a result of the degradation of red blood cells (RBCs) within the hematoma.




Fractures


The clavicle, or collarbone, is the bone most frequently fractured during the birth process. It is often associated with shoulder dystocia or a difficult vertex or breech delivery of infants who are large for gestational age. Crepitus (the coarse crackling sensation produced by the rubbing together of fractured bone fragments) may be felt or heard on examination. A palpable, spongy mass, representing localized edema and hematoma, may also be a sign of a fractured clavicle. The infant may be reluctant to move the arm on the affected side, and the Moro reflex may be asymmetric. Radiographs usually reveal a complete fracture with overriding of the fragments.


Fractures of long bones, such as the femur or the humerus, are sometimes difficult to detect by radiographic examination in infants. Although osteogenesis imperfecta is a rare finding, a newborn infant with a fracture should be assessed for other evidence of this congenital disorder.


Fractures of the neonatal skull are uncommon. The bones, which are less mineralized and more compressible than bones in older infants and children, are separated by membranous seams that allow sufficient alteration in the head contour so that it adjusts to the birth canal during delivery. Skull fractures usually follow a prolonged, difficult delivery or forceps extraction. Most fractures are linear, but some may be visible as depressed indentations that compress or decompress like a ping-pong ball. Management of depressed skull fractures is controversial; many resolve without intervention. Nonsurgical elevation of the indentation using a hand breast pump or vacuum extractor has been reported (Mangurten and Puppala, 2011). Surgery may be required in the presence of bone fragments or signs of increased intracranial pressure (ICP) (Hill, 2008). A similar finding in neonates is craniotabes, which is usually benign or may be associated with prematurity or hydrocephalus (Johnson, 2009). In this condition, the cranial bone(s) move freely on palpation and may easily compress.





Nursing Care Management


Often, no intervention is needed other than maintaining proper body alignment, careful dressing and undressing of the infant, and handling and carrying that support the affected bone. For example, if the infant has a fractured clavicle, it is important to support the upper and lower back rather than pulling the infant up from under the arms. Placing the infant in a side-lying position with the affected side down should also be avoided. Linear skull fractures usually require no treatment. A ping-pong ball–type skull fracture may require decompression by surgical intervention. The infant is carefully observed for signs of neurologic complications. The parents of infants with a fracture of any bone should be involved in caring for the infant during hospitalization as part of discharge planning for care at home.



Paralysis


Facial Paralysis


Pressure on the facial nerve (cranial nerve VII) during delivery may result in injury to that nerve. The primary clinical manifestations are loss of movement on the affected side, such as an inability to completely close the eye, drooping of the corner of the mouth, and absence of wrinkling of the forehead and nasolabial fold (Fig. 9-2). The paralysis is most noticeable when the infant cries. The mouth is drawn to the unaffected side, the wrinkles are deeper on the normal side, and the eye on the involved side remains open.



No medical intervention is necessary. The paralysis usually disappears spontaneously in a few days but may take as long as several months.



Brachial Palsy


image Plexus injury results from forces that alter the normal position and relationship of the arm, shoulder, and neck. Erb palsy (Erb-Duchenne paralysis) is caused by damage to the upper plexus and usually results from stretching or pulling away of the shoulder from the head, as might occur with shoulder dystocia or with a difficult vertex or breech delivery. Other identified risk factors include an infant with birth weight of more than 4000 g (8.8 pounds), a second stage of labor of less than 15 minutes, maternal body mass index greater than 29, a vacuum-assisted extraction, prolonged labor, and a previous history of brachial plexus injury (Hale, Bae, and Waters, 2009; Hudic, Fatusic, Sinanovic, and others, 2006). The less common lower plexus palsy, or Klumpke palsy, results from severe stretching of the upper extremity while the trunk is relatively less mobile.


image Animation—Erb Palsy


The clinical manifestations of Erb palsy are related to the paralysis of the affected extremity and muscles. The arm hangs limp alongside the body while the shoulder and arm are adducted and internally rotated. The elbow is extended, and the forearm is pronated, with the wrist and fingers flexed; a grasp reflex may be present because finger and wrist movement remain normal (Tappero, 2009) (Fig. 9-3). In lower plexus palsy, the muscles of the hand are paralyzed, with consequent wrist drop and relaxed fingers. In a third and more severe form of brachial palsy, the entire arm is paralyzed and hangs limp and motionless at the side. The Moro reflex is absent on the affected side for all forms of brachial palsy.



Treatment of the affected arm is aimed at preventing contractures of the paralyzed muscles and maintaining correct placement of the humeral head within the glenoid fossa of the scapula. Complete recovery from stretched nerves usually takes 3 to 6 months. Full recovery is expected in 88% to 92% of infants (Verklan and Lopez, 2011). However, avulsion of the nerves (complete disconnection of the ganglia from the spinal cord that involves both anterior and posterior roots) results in permanent damage. For injuries that do not improve spontaneously by 3 months, surgical intervention may be needed to relieve pressure on the nerves or to repair the nerves with grafting (Joyner, Soto, and Adam, 2006). In some cases, injection of botulinum toxin A into the pectoralis major muscle may be effective in reducing muscle contractures after birth-related brachial plexus injuries (Price, Ditaranto, Yaylali, and others, 2007).



Phrenic Nerve Paralysis


image Phrenic nerve paralysis results in diaphragmatic paralysis as demonstrated by ultrasonography, which shows paradoxic chest movement and an elevated diaphragm. Initially, radiography may not demonstrate an elevated diaphragm if the neonate is receiving positive-pressure ventilation (Volpe, 2008). The injury sometimes occurs in conjunction with brachial palsy. Respiratory distress is the most common and important sign of injury. Because injury to the phrenic nerve is usually unilateral, the lung on the affected side does not expand, and respiratory efforts are ineffectual. Breathing is primarily thoracic, and cyanosis, tachypnea, or complete respiratory failure may be seen. Pneumonia and atelectasis on the affected side may also occur.


image Animation—Paralyzed Diaphragm



Nursing Care Management


Nursing care of an infant with facial nerve paralysis involves aiding the infant in sucking and helping the mother with feeding techniques. Because part of the mouth cannot close tightly around the nipple, the use of a soft rubber nipple with a large hole may be helpful. The infant may require gavage feeding to prevent aspiration. Breastfeeding is not contraindicated, but the mother will need additional assistance in helping the infant grasp and compress the areolar area.


If the eyelid of the eye on the affected side does not close completely, artificial tears can be instilled daily to prevent drying of the conjunctiva, sclera, and cornea. The eyelid is often taped shut to prevent accidental injury. If eye care is needed at home, the parents are taught the procedure for administering eye drops before the infant is discharged from the nursery (see Chapter 22).


Nursing care of the newborn with brachial palsy is concerned primarily with proper positioning of the affected arm. The affected arm should be gently immobilized on the upper abdomen; passive range-of-motion exercises of the shoulder, wrist, elbow, and fingers are initiated at 7 to 10 days of age (Joyner, Soto, and Adam, 2006). Wrist flexion contractures may be prevented with the use of supportive splints. In dressing the infant, preference is given to the affected arm. Undressing begins with the unaffected arm, and redressing begins with the affected arm to prevent unnecessary manipulation and stress on the paralyzed muscles. Teach parents to use the “football” position when holding the infant and to avoid picking up the child from under the axillae or by pulling on the arms.


The infant with phrenic nerve paralysis requires the same nursing care as any infant with respiratory distress. Mechanical ventilation may be required to prevent further respiratory compromise.


The family’s emotional needs are also an important part of nursing care; the family will need reassurance regarding the neonate’s progress toward an optimal outcome.


Follow-up is also essential because of the extended length of recovery. Parents may wish to contact the Brachial Plexus Palsy Foundation* and visit the website for further information.



Common Problems in the Newborn


Erythema Toxicum Neonatorum


image Erythema toxicum neonatorum, also known as flea-bite dermatitis or newborn rash, is a benign, self-limiting eruption of unknown cause that usually appears within the first 2 days of life. The lesions are firm, 1- to 3-mm, pale yellow or white papules or pustules on an erythematous base; they resemble flea bites. The rash appears most commonly on the face, proximal extremities, trunk, and buttocks, but it may be located anywhere on the body except the palms and soles. The rash is more obvious during crying episodes. There are no systemic manifestations, and successive crops of lesions heal without pigmentation changes. The rash usually lasts about 5 to 7 days. The etiology is unknown. However, a smear of the pustule will show numerous eosinophils and a relative absence of neutrophils. When the diagnosis is questionable, bacterial, fungal, or viral cultures should be obtained. Although no treatment is necessary, parents are usually concerned about the rash and need to be reassured of its benign and transient nature.


image Case Study—Health Problems of the Newborn



Candidiasis


Candidiasis, also known as moniliasis, is not uncommon in newborns. Candida albicans, the usual organism responsible, may cause disease in any organ system. It is a yeastlike fungus (it produces yeast cells and spores) that can be acquired from a maternal vaginal infection during delivery; from person-to-person transmission (especially from poor hand-washing technique); or from contaminated hands, bottles, nipples, or other articles. Mucocutaneous, cutaneous, and disseminated candidal infections are all observed in this age group. Candidiasis is usually a benign disorder in neonates, often confined to the oral and diaper regions. Diaper dermatitis caused by Candida organisms manifests as a moist, erythematous eruption with small white or yellow pebbly pustules. Small areas of skin erosion may also be seen (see Diaper Dermatitis, Chapter 30).



Oral Candidiasis


Oral candidiasis (thrush) is characterized by white, adherent patches on the tongue, palate, and inner aspects of the cheeks (Fig. 9-4). It is often difficult to distinguish from coagulated milk. The infant may refuse to suck because of pain in the mouth.



This condition tends to be acute in newborns and chronic in infants and young children. Thrush appears when the oral flora is altered as a result of antibiotic therapy or poor hand washing by the infant’s caregiver. Although the disorder is usually self-limiting, spontaneous resolution may take as long as 2 months, during which time lesions may spread to the larynx, trachea, bronchi, and lungs and along the gastrointestinal tract. The disease is treated with good hygiene, application of a fungicide, and correction of any underlying disturbance. The source of infection should be treated to prevent reinfection.


Topical application of 1 ml nystatin (Mycostatin) over the surfaces of the oral cavity 4 times a day, or every 6 hours, is usually sufficient to prevent spread of the disease or prolongation of its course. Several other drugs may be used, including amphotericin B (Fungizone), clotrimazole (Lotrimin, Mycelex), fluconazole (Diflucan), or miconazole (Monistat, Micatin) given intravenously, orally, or topically. To prevent relapse, therapy should be continued for at least 2 days after the lesions disappear (Lawrence and Lawrence, 2011). Gentian violet solution may be used in addition to one of the antifungal drugs in chronic cases of oral thrush; however, the former does not treat gastrointestinal Candida infection and may be irritating to the oral mucosa. Some practitioners avoid its use because it is messy, easily stains clothing, and may be irritating to the oral mucosa. Fluconazole is reportedly more effective than nystatin but it does not have Food and Drug Administration approval for use in infants (Su, Gaskie, and Jamieson, 2008).




Nursing Care Management


Nursing care is directed toward preventing spread of the infection and correctly applying the prescribed topical medication. For candidiasis in the diaper area, the caregiver is taught to keep the diaper area clean and to apply the medication to affected areas as prescribed (see also Diaper Dermatitis, Chapter 30). Older infants with candidal diaper dermatitis can introduce the yeast into the mouth from contaminated hands. Placing clothes over the diaper can prevent this cycle of self-infection.


In cases of oral thrush, nystatin is administered after feedings. Distribute the medication over the surface of the oral mucosa and tongue with an applicator or syringe; the remainder of the dose is deposited in the mouth to be swallowed by the infant to treat any gastrointestinal lesions.


In addition to good hygienic care, other measures to control thrush include rinsing the infant’s mouth with plain water after each feeding before applying the medication and boiling reusable nipples and bottles for at least 20 minutes after a thorough washing (spores are heat resistant). If used, pacifiers should be boiled for at least 20 minutes once daily. If the mother is breastfeeding, it is recommended that simultaneous treatment of the infant and mother occur if either is infected (Lawrence and Lawrence, 2011).



Herpes Simplex Virus


Neonatal herpes is one of the most serious viral infections in newborns, with a mortality rate of up to 60% in infants with disseminated disease. Approximately 86% to 90% of herpes simplex transmission occurs during passage through the birth canal (Shet, 2011). The risk of transmission of genital herpes during vaginal birth is estimated to be between 30% and 50% with active primary infection at term (Gardella and Brown, 2011). However, in up to 80% of cases of neonatal herpes simplex virus (HSV) infection, the mother has no history or symptoms of infection at the time of birth, but serologic testing reveals evidence of the herpes virus (Gardella and Brown, 2011).


Neonatal herpes manifests in one of three ways: (1) with skin, eye, and mouth involvement (SEM); (2) as localized central nervous system (CNS) disease; or (3) as disseminated disease involving multiple organs. In skin and eye disease, a rash appears as vesicles or pustules on an erythematous base. Clusters of lesions are common. The lesions ulcerate and crust over rapidly. Most infants with neonatal herpes eventually develop this characteristic rash, but up to 20% of neonates with disseminated disease do not develop a skin rash (Kimberlin, 2007). Ophthalmologic clinical findings include chorioretinitis and microphthalmia; neurologic involvement such as microcephaly and encephalomalacia may also develop (James, Kimberlin, and Whitley, 2009). Disseminated infections may involve virtually every organ system, but the liver, adrenal glands, and lungs are most commonly affected. In HSV meningitis, infants develop multiple lesions of cortical hemorrhagic necrosis. It can occur alone or with oral, eye, or skin lesions. The presenting symptoms, which may occur in the second to fourth weeks of life, include lethargy, poor feeding, irritability, and local or generalized seizures.



Nursing Care Management


Neonates with herpesvirus or suspected infection (as a result of exposure) should be carefully evaluated for clinical manifestations. The absence of skin lesions in the neonate exposed to maternal herpesvirus does not indicate absence of disease. Contact precautions (in addition to standard precautions) should be instituted according to the American Academy of Pediatrics (AAP) and American College of Obstetricians and Gynecologists (ACOG) (2007) guidelines or hospital protocol. It is recommended that swabs of the mouth, nasopharynx, conjunctivae, rectum, and any skin vesicles be obtained from the exposed neonate; in addition, urine, stool, blood, and cerebrospinal fluid (CSF) specimens should be obtained for culture. Therapy with acyclovir and vidarabine is initiated if the culture results are positive or if there is strong suspicion of herpesvirus infection (AAP, Committee on Infectious Diseases, 2009; James, Kimberlin, and Whitley, 2009). High-dose acyclovir (60 mg/kg/day) has been shown to decrease mortality rates in infants with disseminated HSV (James, Kimberlin, and Whitley, 2009).



Birthmarks


Discolorations of the skin are common findings in newborn infants (see discussion on skin assessment of newborns, Chapter 8). Most, such as mongolian spots or telangiectatic nevi, involve no therapy other than reassurance to parents of the benign nature of these discolorations. However, some can be a manifestation of a disease that suggests further examination of the child and other family members (e.g., the multiple light brown café-au-lait spots that often characterize the autosomal dominant hereditary disorder neurofibromatosis and are common findings in Albright syndrome).


Darker or more extensive lesions demand further scrutiny, and excision of the lesion is recommended when feasible. Such lesions include a reddish brown solitary nodule that appears on the face or upper arm and usually represents a spindle and epithelioid cell nevus (juvenile melanoma); a giant pigmented nevus (or bathing trunk nevus), a dark brown to black, irregular plaque that is at risk of transformation to malignant melanoma; and the dark brown or black macules that become more numerous with age (junctional or compound nevi).


Vascular birthmarks may be divided into the following categories: vascular malformations, capillary hemangiomas, and mixed hemangiomas. Vascular stains (malformations) are permanent lesions that are present at birth and are initially flat and erythematous. Any vascular structure, capillary, vein, artery, or lymphatic may be involved. The two most common vascular stains are the transient macular stain (stork bite, salmon patch, or angel kiss) and the port-wine stain, or nevus flammeus. The port-wine lesions are pink, red, or, rarely, purple stains of the skin that thicken, darken, and proportionately enlarge as the child grows (Fig. 9-5, A). The macular stain is most often located on the eyelids, glabella, or nape of the neck and usually fades over several months but may be prominent with crying or environmental temperature changes (Morelli, 2011).



Port-wine stains may also be associated with structural malformations, such as glaucoma or leptomeningeal angiomatosis (tumor of blood or lymph vessels in the pia-arachnoid) (Sturge-Weber syndrome) or bony or muscular overgrowth (Klippel-Trenaunay-Weber syndrome). Children with port-wine stains on the eyelids, forehead, or cheeks should be monitored for these syndromes with periodic ophthalmologic examination, neurologic imaging, and measurement of extremities.


The treatment of choice for port-wine stains is the use of the flashlamp-pumped pulsed-dye laser. A series of treatments is usually needed. The treatments can significantly lighten or completely clear the lesions with almost no scarring or pigment change.


Capillary hemangiomas, sometimes referred to as strawberry hemangiomas, are benign cutaneous tumors that involve only capillaries. These hemangiomas are bright red, rubbery nodules with a rough surface and a well-defined margin (Fig. 9-5, B). Strawberry hemangiomas may not be apparent at birth but may appear within a few weeks and enlarge considerably during the first year of life and then begin to involute spontaneously. It may take 5 to 12 years for complete resolution. As many as 50% of patients may be left with residual findings such as telangiectasia, redundant fatty tissue, or skin atrophy (Alster and Railan, 2006). Cavernous venous hemangiomas involve deeper vessels in the dermis and have a bluish red color and poorly defined margins. These latter forms may be associated with the trapping of platelets (Kasabach-Merritt syndrome) and subsequent thrombocytopenia (Kelly, 2010; Witt, 2009).


Hemangiomas may also occur as part of the PHACE syndrome (Sidbury, 2010):



Although most hemangiomas require no treatment because of their high rate of spontaneous involution, some vision and airway obstruction may necessitate therapy. Systemic propranolol or prednisone may deter further growth. Subcutaneous injections of interferon or vincristine may be required if prednisone therapy and the pulsed-dye laser fail to control a problematic hemangioma; however, the associated side effects may outweigh the benefits of therapy in some cases (Holland and Drolet, 2010).



Nursing Care Management


Birthmarks, especially those on the face, are upsetting to parents. Families need an explanation of the type of lesion, its significance, and possible treatment.* They can benefit from seeing photographs of other infants before and after treatment for port-wine stains or after the passage of time for hemangiomas. Pictures taken to follow the involution process may further help parents gain confidence that progress is taking place.


If laser therapy is performed, the lesion will have a purplish black appearance for 7 to 10 days, after which the blackness fades and gives way to redness with an eventual lightening of the treated area. During the treatment phase, parents are cautioned to avoid any trauma to the lesion or picking at the scab. The child’s fingernails are trimmed as an added precaution. Washing the area gently with water and dabbing it dry is adequate, although in some cases, a topical antibiotic ointment may be used. No salicylates should be taken during the treatment phase because they decrease the effects of the therapy. The child should be kept out of the sun for several weeks and then protected with a sunscreen of at least SPF 25. Complications associated with laser treatment include redness and bruising and, less commonly, hyperpigmentation, hypopigmentation, and atrophic scarring (Alster and Railan, 2006).



Nursing Care of the High-Risk Newborn and Family


Identification of High-Risk Newborns


A high-risk neonate can be defined as a newborn, regardless of gestational age or birth weight, who has a greater than average chance of morbidity or mortality because of conditions or circumstances associated with birth and the adjustment to extrauterine existence. The high-risk period encompasses human growth and development from the time of viability (the gestational age at which survival outside the uterus is believed to be possible, or as early as 23 weeks of gestation) up to 28 days after birth; thus, it includes threats to life and health that occur during the prenatal, perinatal, and postnatal periods.


There has been increased interest in late-preterm infants of 34 to image weeks of gestation who may receive the same treatment as term infants. Late-preterm infants often experience similar morbidities to preterm infants, including respiratory distress, hypoglycemia requiring treatment, temperature instability, poor feeding, jaundice, and discharge delays, as a result of illness. Therefore, assessment and prompt intervention in life-threatening perinatal emergencies often make the difference between a favorable outcome and a lifetime of disability. It is estimated that late-preterm infants represent 70% of the total preterm infant population and that the mortality rate for this group is significantly higher than that of term infants (7.9 vs. 2.4 per 1000 live births, respectively) (Tomashek, Shapiro-Mendoza, Davidoff, and others, 2007). Because late-preterm infants’ birth weights often range from 2000 to 2500 g (4.4–5.5 pounds) and they appear relatively mature compared with smaller preterm infants, they may be cared for in the same manner as healthy term infants while risk factors for late-preterm infants are overlooked. Late-preterm infants are often discharged early from the birth institution and have a significantly higher rate of rehospitalization than term infants (Escobar, Clark, and Greene, 2006). Discussions regarding high-risk infants in this chapter also refer to late-preterm infants who are experiencing a delayed transition to extrauterine life. Nurses in newborn nurseries should be familiar with the characteristics of neonates and recognize the significance of serious deviations from expected observations. When providers can anticipate the need for specialized care and plan for it, the probability of successful outcome is increased.


The Association of Women’s Health, Obstetric and Neonatal Nurses has published the Late Preterm Infant Assessment Guide (Askin, Bakewell-Sachs, Medoff-Cooper, and others, 2007) for the education of perinatal nurses regarding the late-preterm infant’s risk factors and appropriate care and follow-up care.



Classification of High-Risk Newborns


High-risk infants are most often classified according to birth weight, gestational age, and predominant pathophysiologic problems. The more common problems related to physiologic status are closely associated with the state of maturity of the infant and usually involve chemical disturbances (e.g., hypoglycemia, hypocalcemia) or consequences of immature organs and systems (e.g., hyperbilirubinemia, respiratory distress, hypothermia). Because high-risk factors are common to several specialty areas—particularly obstetrics, pediatrics, and neonatology—specific terminology is needed to describe the developmental status of the newborn (Box 9-1).



Box 9-1   Classification of High-Risk Infants



Classification According to Size




Low-birth-weight (LBW) infant—An infant whose birth weight is less than 2500 g (5.5 pounds) regardless of gestational age


Very low–birth-weight (VLBW) infant—An infant whose birth weight is less than 1500 g (3.3 pounds)


Extremely low–birth-weight (ELBW) infant—An infant whose birth weight is less than 1000 g (2.2 pounds)


Appropriate-for-gestational-age (AGA) infant—An infant whose weight falls between the 10th and 90th percentiles on intrauterine growth curves


Small-for-date (SFD) or small-for-gestational-age (SGA) infant—An infant whose rate of intrauterine growth was slowed and whose birth weight falls below the 10th percentile on intrauterine growth curves (see also Fig. 8-1, B)


Intrauterine growth restriction (IUGR)—Found in infants whose intrauterine growth is restricted (sometimes used as a more descriptive term for SGA infants)


Symmetric IUGR—Growth restriction in which the weight, length, and head circumference are all affected


Asymmetric IUGR—Growth restriction in which the head circumference remains within normal parameters while the birth weight falls below the 10th percentile


Large-for-gestational-age (LGA) infant—An infant whose birth weight falls above the 90th percentile on intrauterine growth charts





*Definitions of late-preterm infants vary among experts, but Engle (2006) suggests the above (which corresponds to 239th day to 259th day from the first day of the last menstrual period).


Formerly, weight at birth was considered to reflect a reasonably accurate estimation of gestational age; that is, if an infant’s birth weight exceeded 2500 g (5.5 pounds), the infant was considered to be mature. However, accumulated data have shown that intrauterine growth rates are not the same for all infants and that other factors (e.g., heredity, placental insufficiency, maternal disease) influence intrauterine growth and birth weight. From these data, a more definitive and meaningful classification system that encompasses birth weight, gestational age, and neonatal outcome has been developed. (See Fig. 8-2 for size comparison of newborn infants.)



Care of High-Risk Newborns


Systematic Assessment


A thorough systematic physical assessment is an essential component in the care of high-risk infants (see Nursing Care Guidelines box). Subtle changes in feeding behavior, activity, color, oxygen saturation (SaO2), or vital signs often indicate an underlying problem. Low-birth-weight (LBW) preterm infants, especially very low–birth-weight (VLBW) or extremely low–birth-weight (ELBW) infants, are ill equipped to withstand prolonged physiologic stress and may die within minutes of exhibiting abnormal symptoms if the underlying pathologic process is not corrected. Alert nurses are aware of subtle changes and react promptly to implement interventions that promote optimum functioning in high-risk neonates. Changes in the infant’s status are noted through ongoing observations of the infant’s adaptation to the extrauterine environment.



image Nursing Care Guidelines


Physical Assessment










Skin Assessment




Note any skin lesions or birthmarks.


Describe any discoloration, reddened area, signs of irritation, blisters, abrasions, or denuded areas, especially where monitoring equipment, infusions, or other apparatus come in contact with skin; also check and note any skin preparation used (e.g., skin disinfectants).


Determine texture and turgor of skin—dry, smooth, flaky, peeling, and so on.


Describe any rash, skin lesion, or birthmarks.


Determine whether intravenous infusion catheter is in place and observe for signs of infiltration.


Describe parenteral infusion lines—location, type (arterial, venous, peripheral, umbilical, central, peripheral central venous), type of infusion (medication, saline, dextrose, electrolyte, lipids, total parenteral nutrition), type of infusion pump and rate of flow, type of catheter, and appearance of insertion site.


Observational assessments of high-risk infants are made according to each infant’s acuity; critically ill infants require close observation and assessment of respiratory function, including continuous pulse oximetry, electrolytes, and evaluation of blood gases. Accurate documentation of the infant’s status is an integral component of nursing care. With the aid of continuous, sophisticated cardiopulmonary monitoring, nursing assessments and daily care may be coordinated to allow for minimal handling of the infant (especially VLBW or ELBW infants) to decrease the effects of environmental stress.



Monitoring Physiologic Data


Most neonates under intensive observation are placed in a controlled thermal environment and monitored for heart rate, respiratory activity, and temperature. The monitoring devices are equipped with an alarm system that indicates when the vital signs are above or below preset limits. However, it is essential to check the apical heart rate and compare it with the monitor reading.


Blood pressure (BP) is monitored routinely in sick neonates by either internal or external means. Direct recording with arterial catheters is often used but carries the risks inherent in any procedure in which a catheter is introduced into an artery. BP values gradually increase over the first month of life in preterm and term infants. BP norms vary by gestational age and weight, medications such as corticosteroids, and disease process. One of the primary considerations in the preterm infant is the relationship between systemic BP and the determination of adequate cerebral blood flow. In the neonatal intensive care unit (NICU), frequent laboratory examinations and their interpretation are integral parts of the ongoing assessment of infants’ progress. Accurate intake and output records are kept on all acutely ill infants. An accurate output can be obtained by collecting urine in a plastic urine collection bag specifically made for preterm infants (see Urine Specimens, Chapter 22) or by weighing the diapers, which is the simplest and least traumatic means of measuring urinary output. The preweighed wet diaper is weighed on a gram scale, and the gram weight of the urine is converted directly to milliliters (e.g., 25 g = 25 ml).


Blood examinations are a necessary part of the ongoing assessment and monitoring of the high-risk newborn’s progress. The tests most often performed are blood glucose, bilirubin, calcium, hematocrit, serum electrolytes, and blood gases. Samples may be obtained from the heel; by venipuncture; by arterial puncture; or by an indwelling catheter in an umbilical vein, an umbilical artery, or a peripheral artery (see Atraumatic Care box, p. 210, and Collection of Specimens, Chapter 22).


When numerous blood samples must be drawn, it is important to maintain an accurate record of the amount of blood being removed, especially in ELBW and VLBW infants, who can ill afford to have their blood supply depleted during the acute phase of their illness. There is an increased emphasis on drawing as little blood as possible from high-risk neonates to minimize the depletion of blood volume and avoid blood transfusions and associated complications. To avoid the need for repeated arterial punctures, pulse oximetry, which measures the saturation or percentage of oxygen in the hemoglobin, is typically used. Although used less frequently than pulse oximetry, transcutaneous carbon dioxide (tcPCO2) is monitored in some situations. The nurse notes changes in oxygenation (or other aspects being monitored) associated with handling and adjusts the infant’s care accordingly. The frequency of vital signs is determined by the infant’s acuity level (seriousness of condition) and response to handling.


The nursing process in the care of high-risk newborns and their families is described in the Nursing Process box.


Jan 16, 2017 | Posted by in NURSING | Comments Off on Health Problems of Newborns

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