CHAPTER 14 Environmental Emergencies
I. GENERAL STRATEGY
A. Assessment
1. Primary and secondary assessment/resuscitation (see Chapter 1)
C. Planning and Implementation/Interventions
F. Age-Related Considerations
II. SPECIFIC ENVIRONMENTAL EMERGENCIES
A. Heat-Related Emergencies
Heat-related emergencies occur when the body is no longer able to regulate its body temperature through normal physiologic mechanisms. Thermoregulation occurs through the preoptic anterior hypothalamus. Information about body temperature is sent to the brain by peripheral and central thermoreceptors located in the skin, limb muscles, and spinal cord. The hypothalamus then initiates methods that help the body maintain a normal or tolerable body temperature. The body attempts to maintain a temperature of 98.6°F (37.1°C). When the body is exposed to excessive heat, it will attempt to dissipate the heat by convection, radiation, or evaporation. Drugs, strenuous activity, and high ambient temperatures can increase internal heat production. Factors such as lack of acclimation, restrictive clothing, and high humidity affect the body’s ability to manage excessive heat. As the body’s temperature increases, there is stimulation of the sweat response to initiate evaporative heat loss. This is the body’s primary mechanism of cooling. Sweating not only assists in cooling the body but also may cause loss of body weight, sodium, and potassium. If fluids and electrolytes are not replaced, dehydration can occur. The body also attempts to dissipate heat by shunting blood to the skin. There is an increase in heart rate, stroke volume, and cardiac output. Additionally, the kidneys conserve fluid for evaporation by retaining salt and water. Under normal circumstances, with time to acclimate, these compensatory mechanisms assist the body in sustaining a normal temperature. However, if additional factors contribute to the heat stress, these mechanisms will fail, resulting in a heat-related emergency. People at risk for heat-related emergencies include the young, the elderly, and individuals not acclimated to hot weather. Heat exhaustion and heat stroke represent progressive degrees of heat illness (Table 14-1).
Emergency | Clinical Presentation | Interventions |
---|---|---|
Heat cramps | Brief, intermittent severe muscular cramps in muscles fatigued by heavy work, excessive fatigue, decreased coordination, nausea, vomiting, headache, dizziness | Place patient in cool environment and massage affected muscles; patient should rest until symptoms subside; administer intravenous salt solution: 0.9% normal saline 1000 mL, over 1 to 3 hr or 23.5% saline in 10- to 20-mL increments; or 0.1% saline solution orally; monitor electrolytes and magnesium levels, with replacement as needed |
Heat exhaustion | Dehydration, malaise, weakness, flulike symptoms, thirst, tachycardia, frontal headache, and muscle cramps | Fluid and electrolyte replacement based on serum electrolytes and calculation of body water deficit; spontaneous cooling to 102° F (39 C); acetaminophen (Tylenol) or ibuprofen (Motrin, Advil) may be administered |
Heat stroke | Hot, dry skin, altered level of consciousness, psychotic behavior, delirium, coma, seizures, and severe muscle cramps; temperature ranges from 106 F (41.4° C) to 116 F (47° C); this is a neurologic emergency | Aggressive cooling to 102° F (39° C) within 1 hr; mannitol (Osmitrol) may be administered to decrease intracranial pressure; initiate respiratory and cardiovascular support early |
Heat Stroke (see Table 14-1)
1. Assessment (heat exhaustion/heat stroke)
2. Analysis: differential nursing diagnoses/collaborative problems
3. Planning and implementation/interventions
4. Evaluation and ongoing monitoring (see Appendix B)
B. Cold-Related Emergencies
Frostbite
2. Analysis: differential nursing diagnoses/collaborative problems
3. Planning and implementation/interventions
4. Evaluation and ongoing monitoring (see Appendix B)