Environmental Emergencies

CHAPTER 14 Environmental Emergencies





I. GENERAL STRATEGY



A. Assessment




1. Primary and secondary assessment/resuscitation (see Chapter 1)


2. Focused assessment





3) Psychological/social/environmental factors











3. Diagnostic procedures























F. Age-Related Considerations




1. Pediatrics
















2. Geriatric















II. SPECIFIC ENVIRONMENTAL EMERGENCIES



A. Heat-Related Emergencies


Heat-related emergencies occur when the body is no longer able to regulate its body temperature through normal physiologic mechanisms. Thermoregulation occurs through the preoptic anterior hypothalamus. Information about body temperature is sent to the brain by peripheral and central thermoreceptors located in the skin, limb muscles, and spinal cord. The hypothalamus then initiates methods that help the body maintain a normal or tolerable body temperature. The body attempts to maintain a temperature of 98.6°F (37.1°C). When the body is exposed to excessive heat, it will attempt to dissipate the heat by convection, radiation, or evaporation. Drugs, strenuous activity, and high ambient temperatures can increase internal heat production. Factors such as lack of acclimation, restrictive clothing, and high humidity affect the body’s ability to manage excessive heat. As the body’s temperature increases, there is stimulation of the sweat response to initiate evaporative heat loss. This is the body’s primary mechanism of cooling. Sweating not only assists in cooling the body but also may cause loss of body weight, sodium, and potassium. If fluids and electrolytes are not replaced, dehydration can occur. The body also attempts to dissipate heat by shunting blood to the skin. There is an increase in heart rate, stroke volume, and cardiac output. Additionally, the kidneys conserve fluid for evaporation by retaining salt and water. Under normal circumstances, with time to acclimate, these compensatory mechanisms assist the body in sustaining a normal temperature. However, if additional factors contribute to the heat stress, these mechanisms will fail, resulting in a heat-related emergency. People at risk for heat-related emergencies include the young, the elderly, and individuals not acclimated to hot weather. Heat exhaustion and heat stroke represent progressive degrees of heat illness (Table 14-1).


Table 14-1 HEAT-RELATED EMERGENCIES AND INTERVENTIONS



















Emergency Clinical Presentation Interventions
Heat cramps Brief, intermittent severe muscular cramps in muscles fatigued by heavy work, excessive fatigue, decreased coordination, nausea, vomiting, headache, dizziness Place patient in cool environment and massage affected muscles; patient should rest until symptoms subside; administer intravenous salt solution: 0.9% normal saline 1000 mL, over 1 to 3 hr or 23.5% saline in 10- to 20-mL increments; or 0.1% saline solution orally; monitor electrolytes and magnesium levels, with replacement as needed
Heat exhaustion Dehydration, malaise, weakness, flulike symptoms, thirst, tachycardia, frontal headache, and muscle cramps Fluid and electrolyte replacement based on serum electrolytes and calculation of body water deficit; spontaneous cooling to 102° F (39 C); acetaminophen (Tylenol) or ibuprofen (Motrin, Advil) may be administered
Heat stroke Hot, dry skin, altered level of consciousness, psychotic behavior, delirium, coma, seizures, and severe muscle cramps; temperature ranges from 106 F (41.4° C) to 116 F (47° C); this is a neurologic emergency Aggressive cooling to 102° F (39° C) within 1 hr; mannitol (Osmitrol) may be administered to decrease intracranial pressure; initiate respiratory and cardiovascular support early




Heat Stroke (see Table 14-1)


Heat stroke is a medical emergency. The patient is no longer able to dissipate heat because of failure of the central thermoregulation mechanisms. Morbidity is directly related to the amount of time the patient’s body temperature remains elevated. Exertional heat stroke occurs primarily in young athletes. There is a history of extreme exertion in a hot environment with symptoms occurring over hours. Classic, or nonexertional heat stroke, more commonly occurs during periods of exposure to environmental temperatures exceeding 102.5°F (39.2°C) for a prolonged period of time. Risk factors include age extremes, concurrent use of phenothiazines, and debilitation. There is a lack of acclimation, and the core body temperature rises to greater than 106°F (41°C). The patient’s mental status ranges from confusion to coma, and the skin is hot and dry. Because of fluid loss, the patient is hypotensive and tachycardic. Patients with preexisting cardiovascular disease are prone to developing a hypodynamic state with a low cardiac index (output) and high systemic vascular resistance. Cardiovascular collapse may follow. Treatment includes stabilization of the patient’s airway, breathing, and circulation, along with rapid cooling. There continues to be controversy about which method is best. Cooling methods include the following: removal of clothing, covering the patient with wet sheets, and using a fan to enhance evaporative cooling; providing an ice water bath (conductive cooling); and administering cool fluids. Whatever cooling method is chosen, the patient’s temperature must be closely monitored, and shivering must be controlled. Fluid and electrolyte imbalances must be corrected, and the patient must be monitored for rhabdomyolysis. Clotting studies should be obtained to monitor the patient for the development of disseminated intravascular coagulation (DIC), a potential complication of heat stroke.




1. Assessment (heat exhaustion/heat stroke)








2. Analysis: differential nursing diagnoses/collaborative problems








3. Planning and implementation/interventions


















4. Evaluation and ongoing monitoring (see Appendix B)










B. Cold-Related Emergencies


In a cool environment, the body will attempt to maintain normothermia by conserving heat through vasoconstriction and producing heat through shivering. Risk factors for developing a cold-related emergency include extremes of age, ambient temperature, inappropriate clothing, wet clothing, water/metal contact, wind, and length of exposure. Alcohol, drugs such as phenothiazines, trauma, and illnesses such as diabetes pose additional risks for the development of cold-related emergencies. Cold-related emergencies include frostbite and hypothermia.



Frostbite


Frostbite is true tissue freezing, which is the formation of ice crystals in the tissue. Ninety percent of frostbite cases involve the hands and feet; the cheeks, nose, ears, and penis are also commonly affected. Contributing factors include alcohol intoxication, homelessness, and not being appropriately dressed for the weather. The initial response to cold stress is peripheral vasoconstriction. Cooling increases blood viscosity and decreases capillary perfusion. It results in sludging and thrombosis of the vessels, decreased blood flow, and vascular stasis, which contribute to the development of tissue damage. Subsequently, alterations in vascular permeability lead to edema, progressive dermal ischemia, inflammatory mediator release, neutrophil adhesion, and endothelial damage.


Frostbite may be divided into four degrees, based on the appearance after rewarming: first-degree injury with a central, pale area and surrounding erythema; second-degree injury with blisters surrounded by erythema and edema; third-degree with hemorrhagic blisters and eschar formation; and fourth-degree frostbite with necrosis and tissue loss. Any patient with frostbite must always be evaluated for hypothermia. Treatment includes rewarming of the affected part, pain management, and wound care.




1. Assessment








2. Analysis: differential nursing diagnoses/collaborative problems








3. Planning and implementation/interventions

























4. Evaluation and ongoing monitoring (see Appendix B)





Hypothermia


Hypothermia has been defined as a core body temperature of less than 95°F (35°C). Hypothermia results when the body cannot maintain an adequate temperature. The body produces heat through cellular metabolism, muscle activity, and shivering. Heat is lost through conduction, convection, evaporation, and radiation. The ambient temperature does not have to be very low to cause hypothermia. Hypothermia needs to be rapidly recognized and treated in an effort to avoid life-threatening complications such as apnea, ventricular fibrillation, and acidosis. There are multiple risk factors for hypothermia, including the following: age (pediatric patients because of their inability to shiver and decreased body fat and elderly patients because of the high incidence of cardiovascular disease and decreased body fat); medications, such as phenothiazines and neuromuscular blocking agents, which interfere with the patient’s ability to shiver; alcohol; traumatic injury; shock; and diseases such as diabetes. The treatment of hypothermia focuses on returning the core body temperature to normal and providing supportive care. The method of rewarming is dependent on the severity of hypothermia.



Nov 8, 2016 | Posted by in NURSING | Comments Off on Environmental Emergencies

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