Urinary incontinence

CHAPTER 31 Urinary incontinence


Urinary incontinence is any involuntary loss of urine. It occurs as a result of pathological, anatomical, psychological, or physiological factors that produce obstruction, bladder irritability, or interference with neurological functioning. Environmental factors, such as decreased mobility or inaccessibility of toilet facilities, may also produce periodic incontinence.


Urinary incontinence is a common problem, particularly in older adults. It is so common in older women that some think of it as “normal.” The prevalence in U.S. women is 26% during reproductive years and 30% to 40% in postmenopausal years. In noninstitutionalized elderly women, the prevalence is 15% to 30%, and in men it is 8% to 22%. In elderly persons in nursing homes, the rate rises to almost 50%.


Urinary incontinence in adults is categorized according to the underlying anatomical or physiological impairment—specifically, stress incontinence, urge incontinence (overactive bladder), overflow incontinence, and incontinence from reversible causes.


Stress incontinence is leakage of urine during activities that increase abdominal pressure, such as coughing, sneezing, laughing, or other physical activities. It occurs most often in females and is caused by hypermotility at the base of the bladder and urethra associated with pelvic floor relaxation or intrinsic urethral weakness.


Urge incontinence is an abrupt and strong desire to void with the inability to delay urination and is caused by bladder hyperactivity or hypersensitive bladder. Detrusor muscle overactivity occurs when pathological brain disorders interfere with central inhibitory centers and fail to prevent detrusor muscle contractions.


Overflow incontinence occurs with overdistention of the bladder caused by an underactive or acontractile detrusor muscle; by sphincter-detrusor dyssynergia, which is loss of the synergistic urinary sphincter relaxation that normally occurs with bladder detrusor muscle contraction; or from bladder outlet or urethral obstruction. Sphincter weakness can occur from damage to the urethra or its innervation or from pelvic floor muscle relaxation.


Incontinence from reversible factors originates outside of the lower urinary tract and is caused by mental status impairment, immobility, or medication. Some sources term this functional or transient incontinence.


A final category of incontinence is called mixed incontinence. This occurs when the incontinence is produced as the result of several anatomical, physiological, or functional factors. Involuntary discharge of urine in children is abnormal beyond the age of 4 years for daytime wetting and beyond the age of 6 for nighttime wetting. Daytime wetting constitutes diurnal enuresis; nighttime wetting is known as nocturnal or sleep enuresis. In children, enuresis may be organic or nonorganic; nonorganic enuresis can be primary or secondary. Primary nonorganic enuresis occurs in 75% to 90% of children. This enuresis is defined as wetting that has continued since infancy without an established pattern of dryness. Secondary nonorganic enuresis occurs in 10% to 25% of children and is defined as recurrence of wetting after continence has been established for at least 6 months. The possibility of abnormal urinary anatomy is high in young children who present with urinary tract symptoms.



Diagnostic reasoning: focused history



Adults






Medications


Hypnotic-sedatives, diuretics, anticholinergic agents, adrenergic agents, and calcium channel blockers can cause incontinence. α-Adrenergic agonists and β-adrenergic agonists increase sphincter tone and may cause retention. Anticholinergics, prostaglandin inhibitors, calcium channel blockers, and narcotic analgesics decrease detrusor tone. Diuretics can cause incontinence because of increased production of urine. Central nervous system (CNS) depressants, such as hypnotic-sedatives, can interfere with functional ability.


Table 31-1 lists categories of medications and their mechanism of action in urinary incontinence.


Table 31-1 Medications that Can Cause or Contribute to Urinary Incontinence



































































MEDICATION CATEGORY TYPE OF INCONTINENCE MECHANISM OF ACTION
Anticholinergics Overflow Decreased bladder contractions with retention
Antidepressants Overflow Decreased bladder contractions with retention
Antipsychotics Overflow Decreased bladder contractions with retention
Sedative-hypnotics Overflow Decreased bladder contractions with retention
Antihistamines Overflow Decreased bladder contractions with retention
Narcotics Overflow Decreased bladder contractions with retention
Alcohol Overflow Decreased bladder contractions with retention
Calcium channel blockers Overflow Decreased bladder contractions with retention
β-Adrenergic agonists Overflow Decreased bladder contractions with retention
α-Adrenergic agonists Overflow Sphincter contraction with outflow obstruction
α-Adrenergic antagonists Stress Sphincter relaxation with urinary leakage
Diuretics Urge Contractions stimulated by high urine flow
Caffeine Urge Diuretic effect
Sedative-hypnotics Urge Depressed CNS inhibition of micturition
Alcohol Urge Diuretic effect and depressed CNS inhibition

Adapted from Weiss BD: Diagnostic evaluation of urinary incontinence in geriatric patients, Am Fam Physician 57:2675, 2688, 1998.












Apr 10, 2017 | Posted by in NURSING | Comments Off on Urinary incontinence

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